https://ntp.niehs.nih.gov/go/gmm02abs

Abstract for GMM-2

Toxicity Studies of Acesulfame Potassium in FVB/N-TgN(v-Ha-ras)Led (Tg.AC) Hemizygous Mice and Carcinogenicity Studies of Acesulfame Potassium in B6.129-Trp53tm1Brd (N5) Haploinsufficient Mice (Feed Studies)

CASRN: 55589-62-3
Chemical Formula: C4H4KNO4S
Molecular Weight: 201.25
Synonyms/Common Names: ASK; HOE-095K; 6-methyl-1,2,3-oxathiazine-4(3-H)-one-2,2-dioxide potassium salt
Report Date: October 2005

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Abstract

Acesulfame potassium is an artificial sweetener used throughout the world in food and beverages. Acesulfame potassium was nominated by The Center for Science in the Public Interest because of its widespread use. Male and female Tg.AC hemizygous and p53 haploinsufficient mice were exposed to acesulfame potassium (at least 99% pure) in feed for 9 months. Genetic toxicology studies were conducted in mouse peripheral blood erythrocytes.

9-Month Study in Tg.Ac Hemizygous Mice

Groups of 15 male and 15 female Tg.AC hemizygous mice were fed diets containing 0%, 0.3%, 1%, or 3% acesulfame potassium (equivalent to average daily doses of approximately 420, 1,400, or 4,500 mg acesulfame potassium/kg body weight to males and 520, 1,700, or 5,400 mg/kg to females) for 40 weeks. Exposure to acesulfame potassium had no effect on survival or mean body weights. Feed consumption by the exposed groups was similar to that by the control groups throughout the study. There were no neoplasms or nonneoplastic lesions that were attributed to exposure to acesulfame potassium.

9-Month Study in p53 Haploinsufficient Mice

Groups of 15 male and 15 female p53 haploinsufficient mice were fed diets containing 0%, 0.3%, 1%, or 3% acesulfame potassium (equivalent to average daily doses of approximately 475, 1,500, or 4,700 mg/kg to males and 570, 1,800, or 5,700 mg/kg to females) for 40 weeks. Exposure to acesulfame potassium had no effect on survival or mean body weights. Feed consumption by the exposed groups was similar to that by the control groups throughout the study. There were no neoplasms or nonneoplastic lesions that were attributed to exposure to acesulfame potassium.

Genetic Toxicology

Acesulfame potassium did not increase the frequency of micronucleated erythrocytes in peripheral blood of male or female Tg.AC hemizygous mice administered 0.3% to 3% in dosed feed. A similar study was conducted in p53 haploinsufficient mice, and a significant exposure concentration-related increase in the frequency of micronucleated erythrocytes was noted in males but not females.

Conclusions

Under the conditions of this 9-month feed study, there was no evidence of carcinogenic activity of acesulfame potassium in male or female p53 haploinsufficient mice exposed to 0.3%, 1%, or 3%.

Studies

Summary of the 9-Month Carcinogenesis and Genetic Toxicology Studies of Acesulfame Potassium

 
Male
p53 Haploinsufficient
Mice
Female
p53 Haploinsufficient
Mice

Concentrations in feed

0%, 0.3%, 1%, or 3%
(0, 3,000, 10,000, or 30,000 ppm)
0%, 0.3%, 1%, or 3%

Body weights

Exposed groups similar to the control group Exposed groups similar to the control group

Survival rates

14/15, 15/15, 15/15, 14/15 14/15, 14/15, 14/15, 14/15

Nonneoplastic effects

None None

Neoplastic effects

None None

Level of evidence of carcinogenic activity

No evidence No evidence

Genetic Toxicology

Micronucleated erythrocytes
Mouse peripheral blood in vivo:


Positive in p53 haploinsufficient males; negative in Tg.AC hemizygous males and females and p53 haploinsufficient females